Tissue management is an important aspect of Pressure ulcer healing process. Inflammation is a critical element of TIME framework in the wound care. For the purpose of this research, I have opted to discuss the underlying clinical approaches to inflammation that will help in advancing healing. According to Hoeman, (2008, p.268), the extensive research on the infection cycle of pressure ulcers has provided a predictive timeframe for healing and complete closure of the wound.

For pressure ulcers to heal, inflammation trigger on the body will occur. To avoid dangerous implications on the advancement of wounds, homeostasis need to begin (Kifer, 2012, p.60). Otherwise, possibility of chronic inflammation must be monitored by the patient and the clinician. The phase of blood vessels constriction that will help in creation of clotting through platelets need to be handled with care and seriousness. Such factors as bacterial count and host resistance determine the healing process. In that regard, the pace of proliferation should be monitored so that immediate attention is given upon observed healing delay (Baranoski & Ayello, 2008, p.18).

The clinician need to make consistent assessment of the wound and explain to the patient what changes in the wound color and pain mean. For instance, as the inflammation take effect, the observation of swellings, some heat and redness, and mild pain. However, presence of such features as biofilms should be understood as contributing to delay in healing. According to Maklebust & Sieggreen, (2000, p.28), a progressive wound closure is subject to phagocytosis. In essence, the patient need to keep shifting positioning of the infected part of the pressure to avoid additional pressure and enhance growth factor stimulation. Naturally, the inflammation phase should be assessed for about three days. Any stagnated healing require immediate medical attention from Pressure Ulcers specialist.

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